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Heart disease is the leading killer of both men and women after the age of 45. High cholesterol is responsible for 70% of heart disease. It leads to arterial blockage, heart attacks, hardening of the arteries, blood clots, clogged arteries, and stroke. Over 26 million Americans have high cholesterol. High cholesterol it is not just a disease which effects adults. More and more it is being discovered that our teenagers and even preteens are walking around with this potentially life threatening condition!
With all the bad press these days about cholesterol, It may surprise you to learn that cholesterol is actually an essential part of every cell's structure. It is needed for proper brain and nerve function and is the basis for the manufacture of sex hormones as well.
To understand this apparent contradiction as to how cholesterol can be both "bad" and "good", it is important to understand that cholesterol is manufactured in the liver and transported through the bloodstream to the sites where it is needed. Since It is a fatty substance; and because blood is mainly water; it has to latch on to molecules called lipoproteins in order to travel around successfully.
When we talk about "good" and "bad" cholesterol, we are actually distinguishing between the two different types of lipoproteins cholesterol latches on to:
High density lipoproteins (HDL's) are considered "good cholesterol" because they carry unneeded cholesterol away from the cells and back to the liver, where it is broken down for removal from the body. If everything is functioning as it should, this system remains in balance. However, if there is too much cholesterol for the HDLs to pick up promptly, or if there are not enough HDLs to do the job, cholesterol can form plaque that sticks to artery walls and may eventually cause heart disease
Low density lipoproteins (LDL's) are the major transporters of cholesterol in the bloodstream and are considered "bad cholesterol" because they carry fats out of the liver to the blood vessels. LDLs seem to encourage the deposit of cholesterol in the arteries.
It is also important to distinguish between serum cholesterol and dietary cholesterol. Serum cholesterol is the cholesterol in the bloodstream. Dietary cholesterol is cholesterol that is present in food. While eating foods high in dietary cholesterol can raise serum cholesterol, it is not the only source of serum cholesterol. Indeed, you would have some amount of serum cholesterol even if you never ate any food containing dietary cholesterol because the body produces its own cholesterol.
The National Cholesterol Education Program has set the "safe" level of total serum cholesterol (including both LDL and HDL) at 200 milligrams per deciliter of blood 9mg/dl). A reading above 200 indicates an increased potential for developing heart disease. A level of 200 to 239 is borderline, and levels over 240 are considered to indicate high risk. The normal HDL level for adult men in the United States is 45 to 50 mg/dl, and that for women is 50 to 60 mg/dl.
It is suggested that higher HDL levels, such as 70 or 80 mg/dl, may protect against heart disease. An HDL level under 35 mg/dl is considered risky. So if you have a cholesterol reading of 200, with HDL at 80 and LDL at 120, you are considered at low risk for heart disease. On the other hand, even if you have a total cholesterol level well under 200, if your HDL level is under 35, you would still be considered at increased risk of developing cardiovascular disease. In other words, as your HDL decreases, your potential for heart problems intensifies, even if your total is on the low side.
What is Your Risk for a Heart Attack? Click Here to learn more about the factors which impact your risk of having a heart attack.
Lipitor, Zocor, Provacol and other Statin Drugs are Expensive and Dangerous
You may have learned that you have high cholesterol and are taking medication prescribed by your doctor. Take a minute and read the warnings that accompany this medicine. Read them carefully. The print is small but take your time and learn about the dangers you are facing with these dangerous drugs. You are probably killing yourself while at the same time feeling drowsy, dizzy, depressed, and lacking in energy and your usual vitality. That's because these dangerous toxins are slowly poisoning you and increasing your risk of cancer, heart disease, depression, suicide, and general lack of interest in life.
Click here to read more about the risks of taking statin drugs.
Lower Cholesterol Without Drugs
There are several components to a natural cholesterol reduction program:
1. The single most important thing a person can do to lower their cholesterol level is to eat a healthier diet. Specifically, eliminate and/or cut back on fried foods, fatty foods and foods rich in simple carbohydrates. Unfortunately, changing lifelong eating habits can be very difficult and so many people just won't do it.
2. Exercise is an excellent method for improving circulation and cardiovascular function in general. You can reduce cholesterol levels as well as lower your blood pressure with no change in diet just by walking a half hour a day. Furthermore, because exercise is very effective in reducing stress and because hormones (released during stress) stimulate the liver to produce "bad cholesterol", exercise helps reduce cholesterol levels through its stress-reducing action.
3. Increase your liver's metabolism of cholesterol through the use of the following endogenous (ocurring naturally in the body) dietary supplements: Beta Sitosterol, Beta Glucan, Soy Isoflavones & Chromium Picolinate. Though some exogenous (NOT ocurring naturally in the body) supplements such as Guggu and Policosanol may be of some benefit, exogenous supplements in general will only work for about six months. Beyond that you will NOT get any additional benefit from continuing to take them.
Cholesterol Metabolizer® contains these four proven ingredients together (in the correct amounts to be effective) at a fraction of what they would cost separately.
Beta-Sitosterol is a natural plant oil extracted from soybeans, sugar cane and rice. When taken as a dietary supplement, it significantly lowers both total and LDL serum cholesterol levels in the following distinct ways:
- Beta Sitosterol increases the metabolism or breakdown of cholesterol by the liver. The specific liver enzymes which break down saturated fats become more active in the presence of Beta Sitosterol.
- Beta-Sitosterol inhibits cholesterol production in the liver by acting on specific liver enzymes. The liver actually manufactures more cholesterol than is typically absorbed from food. However, HMG-CoA reductase, an important enzyme for the manufacture of cholesterol in the liver, is broken down rapidly in the presence of beta-sitosterol resulting in lower cholesterol production.
- Beta Sitosterol decreases the absorption of cholesterol in the digestive system. It does this by locking to the fat molecules eaten and by blocking the fat molecule absorption gates in the intestines. The fats and cholesterol are then excreted rather than absorbed. A study in the American Journal of Clinical Nutrition showed a 42% decease in cholesterol absorbed when taking beta-sitosterol before eating scrambled eggs.
- All of the above stated actions of beta sitosterol lead to lower circulating "bad cholesterol" levels in the blood. There are possibly even more benefits to taking beta-sitosterol including aiding in weight loss, protecting the lining of the digestive tract and decreasing the risk of gallstones.
Click Here to read about more about overwhelming evidence that beta sitosterol is an extremely effective way to lower your cholesterol levels.
Beta glucan is a polysaccharide found in oats, barley, yeast and mushrooms. For more than a decade, medical researchers have known of the miraculous powers of beta glucan to lower cholesterol and triglycerides levels and strengthen our immune systems, yet most people have never even heard of it. Most vitamin companies and health food stores don't even sell it. Beta glucan not only lowers total and LDL cholesterol levels, It also increases HDL levels.
Click Here to read about some of the many studies that show beyond a shadow of a doubt that beta glucan is a safe, effective, powerful, and proven way to improve your cholesterol levels.
Isoflavones are found in soybeans, chick peas and other legumes. Soybeans, however have the highest concentration of these powerful compounds. Soy contains many individual isoflavones, but the most beneficial of these plant pigment flavonoids are genistein and daidzein.
The American Heart Nutrition Committee (Circulation, December 2000) advised Americans with high cholesterol to add soy protein to their diets. In that report Dr. Erdman said that numerous studies show that soy isoflavones lower LDL, raise HDL, lower triglycerides and lower total cholesterol levels.
When taken as a supplement, soy isoflavones improve blood profiles significantly. They isoflavones help lower LDL levels, as well as increase HDL levels. Soy isoflavones improve the quality of arteries and are even shown to lower blood pressure. All of these effects have been obtained without any change in diet or exercise.
Click Here to read about some of the many impressive human studies that demonstrate soy Isoflavones value in improving blood lipid profiles.
Chromium is a very important trace element which is often deficient in our refined diets. Chromium Picolinate is thought to be the best source of chromium.
CHROMAX® is the patented and most effective form of chromium picolinate. It has been tested for two decades and found to be safe and effective for cholesterol and triglyceride reduction as well as for lowering the risk of diabetes.
CHROMAX® has been shown to lower cholesterol, not by blocking its production as do toxic drugs such as Mevacor and Pravachol, but by increasing the metabolism of cholesterol. Cholesterol is the body's way of protecting itself from toxins (it is a toxin binder) and is also the necessary precursor of many vital sex and adrenal hormones. Chromium facilitates rather than blocks its metabolism.
Click here to read some of the latest research demonstrating Chromium Picolinate's Cholesterol metabolizing properties.
Achieve Healthy Cholesterol levels with 100% Natural Cholesterol Metabolizer®
Cholesterol Metabolizer® is a unique,100% natural, cholesterol reducing formula which is guaranteed to safely and effectively lower your serum cholesterol level. It is the only product which combines the proven effectiveness of beta sitosterol, beta glucan, soy isoflavones and Chromax (chromium picolinate) combined together in one formula. It is quite simply the most effective alternative to the dangerous, doctor prescribed, Statin cholesterol lowering drugs taken by over 12 million americans.
Diet
Most of us consume foods high in dietary fat. Foods which are fried or are high in animal fats will directly raise blood serum levels of "bad cholesterol"
Foods which are high in simple carbohydrates such as sugars, bread, pasta and sweets indirectly raise "bad cholesterol" levels as they are converted into "bad cholesterol" by the liver. This explains why some vegetarians have high cholesterol.
Age
The blood levels of cholesterol tend to increase as we age--a factor doctors consider when deciding treatment options for patients with certain cholesterol levels.
Weight
People who are overweight are more likely to have high blood cholesterol levels. They also tend to have lower HDL levels. The location of the excess weight also seems to play a role in cholesterol levels. A greater risk of increased cholesterol levels occurs when that extra weight is centered in the abdominal region, as opposed to the legs or buttocks.
Gender
Men tend to have higher LDL levels and lower HDL levels than do women, especially before age 50. After age 50, when women are in their post-menopausal years, decreasing amounts of estrogen are thought to cause the LDL level to rise.
Genetics
Some people are genetically predisposed to having high levels of cholesterol. A variety of minor genetic defects can lead to excessive production of LDLs or a decreased capacity for their removal. This tendency towards high cholesterol levels is often passed on from parents to their children. If your parents have high cholesterol, you need to be tested to see if your cholesterol levels are also elevated.
Disease
Diseases such as diabetes can lower HDL levels, increase triglycerides and accelerate the development of atherosclerosis. High blood pressure, or hypertension, can also hasten the development of atherosclerosis, and some medications used to treat it can increase LDL and triglycerides and decrease HDL levels.
Life style
Factors that negatively affect cholesterol levels also include high levels of stress, which can raise total cholesterol levels, and cigarette smoking, which can lower a person's HDL level as much as 15 percent. On the other hand, strenuous exercise can increase HDL levels and decrease LDL levels. Exercise also can help reduce body weight, which, in turn, can help reduce cholesterol. Recent research has shown that moderate alcohol use (one drink per day for women, two drinks a day for men) can raise HDL cholesterol and therefore reduce the risk of heart attack. Despite such research, it is difficult to recommend the habitual use of alcohol because there are also negative health consequences associated with alcohol use and a high potential for abuse.
A visit to any of the numerous health-oriented forums on the internet will quickly reveal hundreds of posts from dissatisfied statin users, describing an alarming array of side-effects: the most common being extreme fatigue, nausea, gastrointestinal problems, and muscle weakness and pain. Complaints about doctors' inability to link their recent health problems with statin use are frequent. In many instances, users report that they put two-and-two together themselves, stopped taking the drugs, and experienced significant or even complete remission of their symptoms. Frequent side effects are no doubt a major reason why up to 75% of people taking statins discontinue their use.
Of course, defenders of statins are quick to point out the low incidence of adverse effects in controlled, randomized clinical trials as proof of their alleged safety. If you look at these studies closely however you will find that when recruiting for statin clinical trials, researchers carefully screen for, and exclude, a wide range of individuals including women of childbearing age, those with a history of drug or alcohol abuse, poor mental function, heart failure, arrhythmia, and other cardiac conditions, liver and kidney disorders, cancer, "other serious diseases", and "hypersensitivity" to statins. Thus, the disparity between the widespread "real-world" prevalence of side effects from statin use and the low prevalence of side effects in clinical trials is hardly surprising. These trials exclude groups that comprise a significant proportion of the real world population, and can hardly be taken as a realistic barometer for the expected incidence of side effects in the general population.
And even with these strict exclusion criteria, there is evidence to show that the clinical experience with statins has been far from trouble-free. Data for the largest statin trial, the Heart Protection Study (HPS), suggest that the daily 40mg dose of simvastatin used was nowhere near as well tolerated as the authors would have us believe. A substantial number of patients did not enter the trial after a six week run-in before randomization; of the 63,603 potential trial participants who entered the original screening, only 32,145 proceeded to the run-in phase. Of these, 11,609 patients - over one third - dropped out before the official start of the trial.
Regardless of what Statin manufacturing companies would have us believe, the dangers from statin use are very real, as illustrated by the tragic death of Mrs. Elnoisa Calabio. Mrs. Calabio's story was presented at an FDA public hearing in May 2000:
"On October 7, 1999, at the age of 48, registered nurse, wife and mother, Elnoisa Calabio, succumbed to the end stages of irreversible dermatomyositis and interstitial pulmonary fibrosis directly caused by her use of a prescribed cholesterol-lowering medication, simvastatin (Zocor). Mrs. Calabio had no substantial risk factors for heart disease. Her blood pressure was controlled. Her cholesterol was slightly high, but not considered dangerous. Tragically, in her last days she knew that the cholesterol lowering drug her doctor had recommended to extend her life was in fact the cause of her fatal illness."
"On October 7, 1999, at the age of 48, registered nurse, wife and mother, Elnoisa Calabio, succumbed to the end stages of irreversible dermatomyositis and interstitial pulmonary fibrosis directly caused by her use of a prescribed cholesterol-lowering medication, simvastatin (Zocor). Mrs. Calabio had no substantial risk factors for heart disease. Her blood pressure was controlled. Her cholesterol was slightly high, but not considered dangerous. Tragically, in her last days she knew that the cholesterol lowering drug her doctor had recommended to extend her life was in fact the cause of her fatal illness."
Sadly, Mrs Calabio's family is hardly alone in grieving the needless, statin-induced loss of a loved one. In August 2001, pharmaceutical giant Bayer was forced to withdraw Baycol (cerivastatin) from the market, after at least fifty-two deaths had been linked to the drug. Baycol was causing rhabdomyolysis, a condition characterized by severe muscle damage. This rare disorder occurs when a large number of skeletal muscle cells die, subsequently releasing massive amounts of muscle protein into the bloodstream. This muscle protein saturates the kidneys, effectively overwhelming their filtration capacities. Indeed, kidney failure was reportedly a major cause of death amongst the Baycol victims. Baycol is not unique in its ability to damage muscle - all the statins have been shown to produce muscle disorders in susceptible patients, and muscle pain is one of the most common reasons for patients being taken off statin drugs.(33) Researchers recently reported that some patients may suffer muscle deterioration caused by statins while still maintaining normal levels of creatine kinase, the most commonly used indicator of muscle damage.
Statin Drugs Can Cause Liver Damage
Statin drugs can cause abnormalities in liver function, occasionally causing mild hepatitis. In fact people with liver problems can not take Statin drugs at all even if they have very high LDL levels. Between one to three percent of people who start taking statins will have a mild to severe elevation in their liver enzymes. Thus, for about one in 5,000 patients, liver enzymes need to be monitored periodically to make sure the liver still functions reasonably.
Statin drugs can cause abnormalities in liver function, occasionally causing mild hepatitis. In fact people with liver problems can not take Statin drugs at all even if they have very high LDL levels. Between one to three percent of people who start taking statins will have a mild to severe elevation in their liver enzymes. Thus, for about one in 5,000 patients, liver enzymes need to be monitored periodically to make sure the liver still functions reasonably.
Because statins have been linked to liver problems and muscle weakness. Many researchers have concluded that patients at low risk of heart disease should very carefully weight the benefits of using statins against the potential risks.
Statin Drugs Can Cause Nerve Damage
Shortly after statin drugs reached the global market, a Danish physician noticed reports of nerve damage in 14 patients. In 1999, he and his colleagues reported on seven of those cases, then wondered how commonly the side effect, called polyneuropathy, occurs. For this study, the researchers probed medical records of all 460,000 residents of a Danish county. They excluded patients with diabetes, which can cause nerve damage, and they accounted for the effects of any other medicines with neurological side effects.
The new study, reported in May in the journal Neurology but largely overlooked by heart experts, comes as drug manufacturers are working on next-generation cholesterol treatments — and as more baby boomers are urged to take the medications to lower their risk of heart attacks and strokes.
Study co-author David Gaist of the University of Southern Denmark says the findings ought to alert doctors to the possibility of nerve damage among patients on statins but should not discourage their use.
Other experts agree. Sidney Smith, chief science officer for the American Heart Association, called Gaist's study interesting, but pointed out that studies of the drugs' effectiveness in 50,000 people have not shown nerve damage as a significant finding. "Nevertheless, physicians should be aware of symptoms that might be related," he says.
Statin Drugs Can Cause Muscle Damage
According to Dr. Paul S. Phillips, a cardiologist at Scripps Mercy Hospital in San Diego, California, satin drugs are not only very expensive but have many side effects and are potentially very dangerous. Phillips has studied patients in his practice and has concluded that all statins have the potential to cause muscle damage (myopathy), which can lead to a fatal condition called rhabdomyolysis, in which the muscle completely breaks down. Bayer's statin drug Baycol was taken off the US market in August 2001, after the Food and Drug Administration (FDA) received at least 52 reports that people taking Baycol had died of rhabdomyolysis. Another 50 have since died, Phillips said.
He said many of his patients complain of severe muscle aches and fatigue while taking statins, even though they show no signs of muscle damage when their blood is tested for a key enzyme called creatine kinase. High levels indicate trouble. The American Heart Association ( news - web sites) and the American College of Cardiology have recommended that patients be taken off statins if their creatine kinase levels are 10 times normal.
Phillips decided to further study his patients with normal creatine kinase levels. He took samples of their muscle tissue while they were taking statins and also during an 8-week period when they were taken off the drugs. In a paper in the October 1st issue of the Annals of Internal Medicine, he reported that of the first four people studied, all had muscle tissue abnormalities that are rarely seen. These four patients also were very weak in stair-stepping and hip adductor strength tests. When the patients went off statins, they felt better, they were stronger and their tissue samples looked normal again, Phillips said. He is testing more patients and gathering more data, but said he thinks that the currently recommended muscle enzyme test may not detect muscle damage in some people who take statins.
He has started a Web site, www.impostertrial.com, to report updates of his findings, and to seek input from statin-takers who are having muscle aches.
Statin drugs deplete essential CoQ10 reserves in the body
Coenzyme Q10 — also called ubiquinone, which means "occurring everywhere" -- plays an important role in the manufacture of ATP, the fuel that runs cellular processes. In addition to this fundamental role in energy production, CoQ10 acts as a potent antioxidant. Though it is present in every cell in your body, it is especially concentrated in the very active cells of your heart. Not surprisingly, CoQ10 is extremely important for cardiovascular health, with high levels being found in healthy heart tissue.
Statins have been shown to deplete the body of Coenzyme Q10. Statins block enzyme pathways involved in the production of cholesterol, thereby lowering cholesterol levels. But the same enzymes that are involved in the production of cholesterol are also required for the production of coenzyme Q10; not surprisingly, lower cholesterol levels in statin users are accompanied by reduced levels of CoQ10.
Depriving the heart of CoQ10 is like removing a spark plug from your engine -- it just won't work. Low levels of CoQ10 are implicated in virtually all cardiovascular diseases, including angina, hypertension, cardiomyopathy and congestive heart failure.
Ironically, while statins are taken to reduce the risk of atherosclerotic heart disease, their CoQ10-robbing effects have been linked to an increased risk of congestive heart failure. Figures from the National Center for Health Statistics show that since the early nineties - when statin drugs began hitting pharmacy shelves - the incidence of congestive heart failure has risen sharply.(37) CHF, in fact, is the fastest growing cardiovascular disorder in the United States. Sadly, there is no cure for CHF short of a heart transplant. Peter H. Langsjoen, MD, a foremost authority on the use of coenzyme Q10 in the treatment of heart disease, has little doubt as to the culprit behind this sharp rise in CHF:
"In my practice of 17 years in Tyler, Texas, I have seen a frightening increase in heart failure secondary to statin usage, "statin cardiomyopathy". Over the past five years, statins have become more potent, are being prescribed in higher doses, and are being used with reckless abandon in the elderly and in patients with "normal" cholesterol levels. We are in the midst of a CHF epidemic in the US with a dramatic increase over the past decade. Are we causing this epidemic through our zealous use of statins? In large part I think the answer is yes."
While CoQ10 is not only critically important for healthy cardiovascular function; the brain is also highly vulnerable to CoQ10 deficiencies. When healthy young men were given either statin drugs or a placebo, those taking lovastatin displayed significant deterioration in cognitive function after only three weeks of treatment.(37) Brian Vonk, M.D., of The Optimal Wellness Center, reported recently that, in his own experience and that of his colleagues, "É statins cause depression or loss of motivation in the majority of patients, probably due to alteration of cholesterol metabolism in the brain. As a result, many of these patients are also on [antidepressant] drugs (e.g. Zoloft, Paxil, Prozac)."
Since the introduction of Statin drugs in 1987, Merck has known that statins deplete CoQ10 reserves, and knew that this could contribute to heart disease. In 1990, Merck sought and received a patent for Mevacor and other statin drugs formulated with up to 1,000 mg of coenzyme Q10 to prevent or alleviate cardiomyopathy, a serious condition that can cause congestive heart failure. Merck however, has not brought these combination products to market, nor have they educated physicians on the importance of supplementing CoQ10 to offset the dangers of these drugs to the heart. Because they hold the patent, other drug companies are prevented from coming out with a statin/CoQ10 product.
Statins and Cancer
In 1996 the Journal of the American Medical Association published an extensive review of the research studying the link between cholesterol-lowering drugs and cancer. The authors, Dr Thomas Newman and Dr. Stephen Hulley, stated: "All members of the two most popular classes of lipid-lowering drugs (the fibrates and the statins) cause cancer in rodents, in some cases at levels of animal exposure close to those prescribed to humans." In light of their findings, the authors recommended that: "lipid-lowering drug treatment, especially with the fibrates and statins, should be avoided except in patients at high short-term risk of coronary heart disease."
Newman and Hulley's recommendation has been all but ignored. Statins are being recommended and prescribed, not just to people at high short-term risk, but to perfectly healthy people who show no clinical manifestations of CHD whatsoever, except for the non-disease of hypercholesterolemia. Even children with "elevated" cholesterol levels are being urged to commence statin therapy.
Drug companies and health authorities repeatedly assure us that statins are wonderful low-risk drugs that are well tolerated in most people. They claim that clinical trials have shown no increase in cancer incidence with statin use, but the longest of these studies ran for only 6 years (excepting the EXCEL trial, which showed an increase in total mortality after 1 year of lovastatin use, and for which no subsequent mortality data has ever been released(44)). Cancer is a chronic disease that may take decades to manifest itself as a life-threatening illness - can we really conclude from trials lasting five to six years that statins are safe for lifetime use? Even heavy smokers are highly unlikely to develop cancer within six years of taking their first puff; most continue for decades before they come to realize the true value of those little warnings adorning cigarette packets.
Because rodent studies routinely use far higher dosages of drugs than those prescribed to humans, some have questioned the relevance of Newman and Hulley's findings. In many studies, rodents have been shown to eliminate drugs much faster than humans, necessitating higher dosages to maintain constant blood levels of the drug. The authors noted, however, that when the drug exposure was considered in terms of blood levels, carcinogenicity occurred at levels close to those seen in humans. In the same journal in which this review appeared, a commentary critical of Hulley and Newman claimed that higher dosages used in rodents placed inordinate stress on their gastrointestinal tracts, and that most of the cancers seen in rodent studies were malignancies of the gastrointestinal tract and liver.(45) Given that gastrointestinal distress and liver toxicity are among the most frequently reported side effects in patients prescribed statins, this proffered explanation provides little reassurance.
Cancer Incidence In Human Statin Trials: A Closer Look.
The claim that statin trials have not shown any increase in cancer is disputed by the recent PROSPER trial, which found a 25% increase in newly diagnosed cancers among elderly individuals treated with pravastatin. While there were 20 less deaths from CHD and stroke in the treatment group, 24 more deaths from cancer were observed, and, in an ominous confirmation of animal findings, one of the highest increases was observed for gastrointestinal cancers.(46) The PROSPER authors dismissed these findings by referring to a pooled analysis they performed of eight statin trials that lasted three or more years, which showed no statistically significant difference in cancer incidence between the placebo and statin groups (6.9% versus 7.1%, respectively). However, most of these trials involved younger subjects. Because cancer risk increases with age, such a comparison bears little relevance to the PROSPER results. Due to their heightened risk, elderly subjects may act as a far more sensitive barometer to any cancer-promoting capacity possessed by statins.
Furthermore, as researcher Uffe Ravnskov, M.D., PhD., recently pointed out, the PROSPER researchers' analysis did not include skin cancer.(47) Considering the relatively short-term nature of statin trials, it is the incidence of such an easily detectable, superficial cancer that would provide the strongest clue as to the future cancer-causing potential of statins. Only two of the statin trials have reported skin cancer incidence; the 4S and HPS simvastatin trials. Increases in skin cancer were noted in both.
In the CARE trial, breast cancer, another readily detectable malignancy, developed in 12 women from the treatment group but in only one of the control individuals - a highly significant difference.(50) Breast cancer was also the malignancy for which the greatest increase was noted in the PROSPER trial. The possibility that statins will lead to future increases in cancer incidence cannot be flippantly dismissed.
To maintain their lipid-lowering effects, statins must be administered on a life-long basis. The reports of carcinogenicity from rodent studies and increases in superficial cancers noted in human trials warrant extreme caution. Given the complete lack of data on the effects of decades of statin administration, users can consider themselves part of a mass experiment in progress, the outcome of which is largely unknown.
Warnings for statin use to be limited to high-risk patients - where dramatically shortened life expectancies may override any concerns about long-term side-effects -have been completely overshadowed by the relentless promotional efforts of drug companies and enthusiastic endorsements from health authorities who are besides themselves at finally having clinical data that, on the surface, appears to support the lipid hypothesis.
Are we witnessing the unfolding of another officially-endorsed health disaster? Only time will tell. For those who do not want to find out the hard way, there are numerous non-drug measures that can help alleviate the risk of CHD. Randomized trials involving increases in fish/fish oil and/or fruit and vegetable consumption have produced risk reductions in mortality similar, and in some instances superior, to those seen in statin trials;(51-57) these safe, natural food items, as well as exercise, stress reduction, sound sleep, and a low glycemic load diet (i.e.., a low-to-moderate carbohydrate diet comprised of unrefined low glycemic foods), would be a far more judicious preventive alternative for those with no clinical signs of CHD.
Statin drugs cost a bundle and are dangerous
They can cause liver, muscle, nerve damage and possibly cancer. If you're taking a statin drug (Zocor, Provacol, etc.), don't wait for your doctor to warn you of the substantial risks. Consider this fact: in the last 15 years (roughly the time that statins have been on the market), the incidence of congestive heart failure has tripled.
For more information visit Naturalnews.com and Dailymail.co.uk
Cholesterol Metabolizer® is a safe inexpensive alternative and it works better for most people.
If there was only one supplement you could take to reduce your cholesterol it should be beta-sitosterol taken in 300-600 mg doses every day. Beta-sitosterol is the most studied, most proven, most effective suplement to lower total and LDL cholesterol. The studies on this in the medical journals actually go back 50 years yet most people have never even heard of it.
Beta-sitosterol is phytosterol or plant alcohol that is literally in every vegetable we eat. We already eat this every day but we just don't get enough of it. The typical American is estimated to eat only 200-400 mg a day while vegetarians probably eat about twice this much. This is surely one of the many reasons vegetarians are healthier and live longer. Actually the term beta-sitosterol in commerce refers to the natural combination of beta-sitosterol, stigmasterol, campesterol and brassicasterol as this is how they are made by nature in plants. There are no magic foods with high levels of phytosterols, but they can be inexpensively extracted from sugar cane pulp, soybeans and pine oil.
Upjohn Pharmaceuticals tried to make a prescription analog (chemical relative) of it decades ago for lowering cholesterol but did not succeed - the natural molecule works best. The scientific community has been well aware of it and clinics around the world have done extensive studies on both humans and animals including gall bladder, bile and liver functions since these are all part of the cholesterol metabolism. The major mechanism this seems to be effective is simply by preventing the dietary cholesterol from being absorbed in the intestines where fat is digested. Another way this seems to work is by increasing the flow of bile acids, which binds the cholesterol in the digestive track and excretes it in the feces.
A good study was done at the Wageningen Agricultural Institute in the Netherlands, the same clinic that did so much good research on trans fatty acids (Am. J. Clin. Nutr. 72, 2000, p. 1510-5). They gave men and women a margarine containing plant sterols and got very significant reductions in cholesterol as well as lower LDL levels in only three weeks. Why a clinic would give margarine to people after studying the negative effects of hydrogenated oils is another matter. Again, these were healthy subjects with normal cholesterol levels, yet they still got great benefits very quickly with no change in diet or exercise.
At Uppsala University in Sweden (Eur. Heart J. Supp. 1, 1999, p. S80-S90) the doctors wanted to give the volunteers the phytosterols in conjunction with a cholesterol lowering diet to see the results of a more comprehensive lifestyle program. The results were really impressive in that the men and women lowered total cholesterol a full 15% and LDL cholesterol a full 19% in less than a month. The shows the very dramatic results you can get with just adding some reasonable dietary changes even without any exercise program at all.
At the University of British Columbia at their St. Paul's Hospital (American Journal of Medicine 107 (1999) p. 588-94) a very impressive review was done complete with 86 references of using plant sterols to lower total cholesterol and LDL since 1951. They said of the recent research, " In 16 recently published human studies that used phytosterols to decrease plasma cholesterol levels in a total of 590 subjects, phytosterol therapy was accompanied by an average 10% decrease in total cholesterol and 13% decrease in LDL cholesterol levels." They found this worked best in high-fat diets; the worse the diet the more results the researchers got.This is the best review to date and should convince anyone of the effectiveness of sterols over drugs.
At McGill University in Montreal (Can. J. Physiol. Pharmacol. 75, 1997, p. 217-27) doctors did a review of the literature on beta-sitosterol and cholesterol metabolism. They researched in detail 18 major studies that used sitosterols to lower cholesterol and triglycerides. They concluded, "addition to diet of phytosterols represents an effective means of improving circulating lipid profiles to reduce risk of coronary heart disease." This study came complete with forty high quality references and left no doubt about the effectiveness of phytosterols on humans. Also at McGill University (Metabolism Clinic Experiments 47, 1998, p. 751-6) patients on a fixed diet were given sterols from pine oil for a mere ten days in a strict, randomized crossover study. This was not a low fat or low cholesterol diet at all. They successfully lowered both their total cholesterol and LDL levels in this short term placebo controlled experiment. They concluded, these results demonstrate the short term efficacy of pine oil plant sterols as cholesterol lowering agents"
A very interesting study was done at the Center for Human Nutrition in France (Ann. Nutr. Metab. 39, 1995, p. 291-5) in that healthy people with normal cholesterol levels were given beta- sitosterol to see if their normal levels could be lowered even further. We always think of studies as using unhealthy people with pathological cholesterol levels given supplements to make them normal again. Amazingly enough the healthy people lowered their normal cholesterol levels even more with no change in diet or exercise. In fact, they were a full 10% lower in only a month. This kind of effect is really fascinating. They said, "The present results may be of great interest in the prevention of high cholesterol diet-associated risks, especially in the prevention of cardiovascular diseases. Since beta-sitosterol was so effective for people who didn't even need it, think what it will do for those people who do need to lower their blood lipids. They concluded, "These findings suggest that a significant lowering of plasma total and LDL cholesterol can be effected by a modest dietary intake of soybean phytosterols"
The University of Helsinki took a big interest in lowering cholesterol with plant sterol therapy back in 1988. The first study (Clinical Chimica Acta 178, p. 41-9) studied familial (genetic) hypercholesteremia. The higher the sterol levels they found in the patients blood the more cholesterol was excreted rather than absorbed. The second study was in 1989 (Metabolism Clinical Experiments 38, p. 136-40). Men were studied again for blood levels of sterols and they found the higher the levels the more cholesterol was excreted successfully. The third study in 1994 (American Journal of Clinical Nutrition 59, p. 1338-46) studied vegetarians who eat twice as many plant sterols as normal people. They showed one reason vegetarians have lower cholesterol levels besides the food they eat is the efficiency of their cholesterol excretion due to their intakes of plant sterols. In the last study in 1999 (Current Opinion Lipidology 10, p. 9-14) they said, "Plant sterols may be useful for the treatment of hyper-cholesteremia they may have a potent cholesterol lowering effect as shown in normal and hypercholesteremic men and women with and without coronary heart disease and diabetes mellitus"
At the University of Kagawa in Tokyo two studies were done. The first was done on healthy young men who were given plant sterols for only five days. In this short time their cholesterol levels fell measurably (Joshi Eiyo Daigaku Kiyo 14, 1983, p. 165-72). The second study was done on healthy young women (same journal 15, 1984 p. 11-18) again giving them plant sterols for only five days. "Administration of phytosterol (mainly sitosterol) increased the output of fecal cholesterol." These were all healthy young Japanese people eating a traditional low-fat diet who did not have a cholesterol problem to begin with, yet they received measurable results in only five days.
At the University of California in San Diego men were isolated in a hospital ward and fed 500 mg of cholesterol and then beta-sitosterol supplements (American Journal of Clinical Nutrition 35, 1982, p. 697-700). This resulted in a 42% decrease in cholesterol absorption in the intestines. They said, "Evidently, the judicious addition of beta-sitosterol to meals containing cholesterol rich foods will result in a decrease in cholesterol absorption with a consequent decrease in plasma cholesterol"
The research is so extensive and wide ranging over the last 30 years that it is hard to find and count all the studies. How something so studied, proven, effective and well known to the scientific and medical community has stayed outside of public knowledge is hard to believe.
Many studies have been done in other areas of illness that suggest beta-sitosterol may have great potential in other areas such as prostate disease, diabetes, blood clotting, ulcers, cancer prevention, tumors, immunity, inflammation and other conditions. These studies have been conducted at such institutions as the State University of New York, National Institutes of Health, University of Japan, University of Valencia, University of Stellengbosch and other prestigious clinics who are willing to study an inexpensive natural plant extract that cannot be patented or sold be prescription. You will see more research and more benefits for beta-sitosterol every year.
Beta glucan is a polysaccharide found in oats, barley, yeast and mushrooms. The miraculous powers of beta glucan to lower cholesterol and triglycerides and strengthen our immune systems have been known about for more than a decade now but until recently, it has been all but impossible to economically extract it from even inexpensive oats and leftover beer brewing yeast. Finally, in the year 2000 technology had advanced to the point where beta glucan can be extracted very inexpensively. At the University of Hamburg in Germany it was shown that all 1,3 configuration beta glucans have the same biological potency whether they are derived from oats or yeast -the two major sources (Carbohydrate Research 297, 1997, p. 135-43). They said, "All glucans investigated, regardless of molar mass and solution structure, stimulate the investigated immunological measures" Just so you will know, yeast and mushrooms are 1,3/1,6 arrangements and oat and barley are 1,3/1,4 structural arrangements, but they are all basically true 1,3 beta glucans.
Actually beta glucan is probably the most powerful immunity enhancer known to science regardless of cost. There are many studies on animals and humans, showing the great value it has to strengthen our immune systems and even the potential to help against tumors and cancer growth. At the University of Saskatchewan in Canada (Microbiol. Immun. 41, 1997, p. 991-8) researchers showed the power to stimulate the immune system. Other studies have found such potential uses as fighting infections, improving intestinal flora, irritable bowel syndrome, diabetic conditions, ulcers and digestion. This, however, is a website on cholesterol so that is what we'll stress. There are many, many studies on blood lipids so we'll just talk about some of the more interesting of the human studies.
At Harvard Medical School in Massachusetts (Crti. Rev. Food Sci. Nutr. 39, 1999, p. 189-202) doctors found that both oat and yeast beta glucans lowered serum cholesterol levels. They did this by simply adding beta glucan to the diets of the people they studied. Notice that there is no use of drugs here and this comes from Harvard Medical School where they are traditionally concerned with prescription drugs and not natural plant supplements. In their words, "In addition to decreasing the intake of total fat, saturated fat and dietary cholesterol, blood serum cholesterol can be further decreased by dietary fiber, especially from sources rich in beta glucan such as oats and yeast".
At the University of Syracuse in New York seventy-one men and women with high cholesterol were given various combinations of low fat diets or regular diets with and without oat beta glucan. In a matter of weeks total cholesterol levels were reduced as much as 17% (Journal of the American Dietary Association 90, 1990, p. 223-9). Their high-density cholesterol levels were also increased significantly. This shows the benefit of making better food choices along with taking effective supplements since the people who ate the low fat diet while taking the oat supplement got the best results.
At the University of Massachusetts (Am. J. Clin. Nutr. 70, 1999, p. 208-12) researchers found that giving obese men with high cholesterol levels yeast derived beta glucan lowered both their total and LDL levels by a full 8% with no change in diet. They summarized the study, "Thus, the yeast derived beta glucan fiber lowered the total cholesterol concentrations and was well tolerated." As usual any side effects were positive in nature.
At the U. S. Human Nutrition Research Center in Maryland (J. Nutr. Biochem. 8, 1997, p. 497-501) people were given oat extracts high in beta glucan content and lowered their cholesterol levels with no changes in diet or exercise. They also found out that other metabolic conditions improved, so new benefits of beta glucan are always being discovered.
Again at the Human Nutrition Research Center (J. Am. Coll. Nutr. 16, 1997, p. 46-51) men and women with high blood lipid levels were given oat extracts high in beta glucan. After only five weeks the groups were switched and those getting the beta glucan received only the usual American diet. Both total cholesterol and LDL levels decreased significantly. In their words, "A significant dose response due to beta glucan concentration in the oat extract was observed in the total cholesterol levels." Thorough studies like this in real people at the most prestigious research centers in the world leave no doubt about the power of beta glucan to lower blood fats.
At Industrial Research Limited in New Zealand (Carbo. Polymers 29, 1996, p. 7-10) researchers used barley derived beta glucan to try and understand the actual metabolic mechanisms by which it lowered blood fats. They first discovered that it increased the secretion of bile acids from the gall bladder. By using highly sophisticated NMR spectroscopy techniques they found the situa-tion to be more complicated than the mere enhanced gall bladder activity. We are more concerned with the practical matters that beta glucan actually works rather than the how and why of it all.
At the University of Lund in Sweden (Ann. Nutr. Metab. 43, 1999, p. 301-9) 66 mildly hypercholesterolemic men were given oat milk in their diet high in beta glucan content for five weeks. This was a classic double blind study where half the men received rice milk with no beta glucan. Of course the men getting the oat milk lowered their total cholesterol and they said, "It is concluded that oat milk has cholesterol reducing properties"
You can see from studies like these there is no doubt that beta glucan is a safe, effective, powerful, proven and inexpensive way to lower your cholesterol levels, yet most people have never even heard of it. Most vitamin companies don't even sell it and it can be difficult to find a reliable, strong, inexpensive brand in your drug store or even in your health food store. People keep taking dangerous, expensive, prescription cholesterol-lowering drugs when then can use natural remedies like beta glucan.
In addition to the benefits we've just covered, it is important to take this because it may well be the most important immune enhancer known to science. Beta glucan strengthens our immune systems so we have optimum healing power in our body and fight off infections of all kinds whether bacteria, fungi or viruses.
You should understand that it is very difficult to study human beings for immune function. Animal studies are used because obviously you can't infect humans with deadly microorganisms and then give half of them beta glucan and see who lives and who doesn't. Animal studies have shown results for such conditions infections of many types, tumors, diabetes, intestinal function and ulcers.
The animal studies have been done for the last ten years in clinics worldwide and published in the major medical journals but beta glucan has only become economically available to consumers in the last few years.
At the University of Saskatchewan beta glucan protected mice from deadly injections of Staphalococcus aureus. In another study nice were injected with equally deadly Eimeria vermiformis but beta glucan protected them. And yet in a third study mice were given the toxic drug dexamethasone and then injected with the deadly Eimeria virus. Even after their immune systems were impaired by the drug the beta glucan protected them. At SRI International the Euglena gracilis virus was injected into various test animals but beta glucan stopped them from dying. At the University of Kansas pigs were given deadly Staphalococcus suis and beta glucan saved them. The doctors there did an in-depth study of various immune system markers to see how it worked.
At the Mayo Clinic lung cancer in mice was reduced by beta glucan. At Tokyo College doctors found strong anti-tumor properties for beta glucan. At Tokyo University doctors found anticancer activity in mice when given beta glucan and suggested they be used as, "biological response modifiers in cancer patients." At Wuhan University they found powerful anti-tumor activity in mice given beta glucan. At the University of Louisville they found the anti-tumor effect of beta glucan was largely due to enhancing beneficial natural killer (NK) cells.
Numerous reports indicate that, because soy is high in isoflavones, it can prevent illness and promote good health.
Isoflavones are a class of phytochemicals, which are compounds found only in plants (phyto means plant). They are also a type of phytoestrogen, or plant hormone, that resembles human estrogen in chemical structure yet are weaker. By mimicking human estrogen at certain sites in the body, isoflavones provide many health benefits that help you to avoid disease.
Isoflavones are found in soybeans, chick peas and other legumes. However, soybeans are unique because they have the highest concentration of these powerful compounds. Soy contains many individual isoflavones, but the most beneficial are genistein and daidzein.
Isoflavones show tremendous potential to fight disease on several fronts. They have been shown to help prevent the buildup of arterial plaque, which reduces the risk of coronary heart disease and stroke. Isoflavones may help reduce breast cancer by blocking the cancer-causing effects of human estrogen. They may also prevent prostate cancer by hindering cell growth. Isoflavones can fight osteoporosis by stimulating bone formation and inhibiting bone resorption. They may even relieve some menopausal symptoms as well.
Soy isoflavones have antioxidant properties which protect the cardiovascular system from oxidation of LDL (the bad) cholesterol. Oxidized LDL cholesterol accumulates in the arteries as patches of fatty buildup which blocks the flow of blood, resulting in atherosclerosis. Genistein inhibits the growth of cells that form this artery clogging plaque. Arteries damaged by atherosclerosis usually form blood clots. This can lead to a heart attack if the clot goes to the heart, or a stroke if it goes to the brain.
Being a weak form of estrogen, isoflavones can compete at estrogen receptor sites, blocking the stronger version naturally produced by the body from exerting its full effect. Since high blood levels of estrogen are an established risk factor for breast cancer; weaker forms of estrogen may provide protection against this disease. Genistein has been found to hinder breast cancer as well as prostate cancer. Results from a new University of California study show that genistein slowed prostate cancer growth and caused prostate cancer cells to die. It acts against cancer cells in a way similar to many common cancer-treating drugs.
Isoflavones also play an important role in protecting and maintaining strong and healthy bones. Evidence shows that genistein and daidzein prevent bones from breaking down. Independent studies conducted at the University of Illinois and the University of Hong Kong concluded that consuming soy isoflavones can increase bone mineral content and bone density. Another study at the University of Texas suggested that isoflavones may also stimulate bone formation. By preserving bone health, increasing bone mass and inducing bone turnover, researchers noted the potential role of soy isoflavones in preventing, and possibly even reversing, the effects of osteoporosis.
The North American Menopause Society suggests that soy isoflavones can also be a natural alternative to estrogen replacement therapy for relief of mild menopausal symptoms. It may help offset the drop in estrogen and regulate its fluctuations that occur at menopause. Many women have reported a reduction in their hot flashes and night sweats when they regularly consume soy foods, like tempeh or tofu.
All these findings suggest eating soy foods, natural sources of isoflavones, can protect and enhance your overall health. Isoflavones work together with soy protein in fighting disease. Studies show that isoflavones account for approximately three-fourths of soy's protection, while its protein is responsible for about one-fourth. The best way to consume isoflavones is in food form, so that you can benefit from all of soy's nutrients and beneficial compounds. The highest amounts of isoflavones and soy protein are found in tempeh, whole soybeans (like edamame), textured soy protein, soynuts, tofu and soymilk. Researchers recommend consuming at least one to two servings a day. A serving is equal to 1 ounce of soynuts; 4 ounces of tempeh, textured soy protein (cooked), or edamame; or 8 ounces of soymilk.
For those new to soy, I recommend slowly adding it to your diet, until you develop a taste for it. In spaghetti sauces, replace ground beef with textured soy protein. Use tofu instead of ricotta cheese in lasagna, or make herb dips with it in a food processor. Use soymilk to cream soups or make smoothies. People on the run can always eat soynuts. Tempeh is one of the easiest soy foods prepare. To make a grilled tempeh sandwich, just cut it into slices, sprinkle on some soy sauce, saute with sliced onions and pile it on some bread. Remember, you will only continue to eat healthy foods if they taste good. So, experiment and have fun trying out new ways to enjoy soy.
Gilbert R. Kaats, PhD, Samuel C. Keith, John A. Wise, PhD, Dennis Pullin, MS, and William G. Squires, Jr., PhD. "Effects of baseline total cholesterol levels on diet and exercise interventions," Journal of the American Nutraceutical Association 2(1): 42-49, 1999
The authors conducted two concomitant studies that examined the effects of a behavior modification plan (BMP) that included the use of nutritional supplements (one group received 400 mcg of chromium as chromium picolinate [CP]) and which collected pre- and post-study serum cholesterol measurements. The results from the CP-supplemented group indicate that "chromium picolinate can facilitate reductions in triglycerides (TC) and LDL serum cholesterol, especially in people whose baseline TC levels were above 200 mg/dL."
Raymond I. Press, MD, Jack Geller, MD, and Gary Evans, PhD. "The effect of chromium picolinate on serum cholesterol and apolipoprotein fractions in human subjects, "The Western Journal of Medicine 152:41-45,1990.
In this study, 28 people were given either 200 mcg of chromium as chromium picolinate (CP) or a placebo, daily, for 42 days in this double-blind crossover study. In this trial, in the patients who supplemented with CP, four of the six most important serum lipids (circulating blood fats) were beneficially altered during the test period. Specifically, levels of total cholesterol, LDL (or "bad") cholesterol and its transport protein, apolipoprotein B, were all beneficially decreased, while levels of the transport protein for HDL (or "good") cholesterol, apolipoprotein A, were beneficially increased. The authors say, "Because each of these variables is related [ƒ] to the development of coronary artery disease, chromium picolinate is an excellent agent to consider in the treatment and prevention of hyperlipidemia."
How Does Cholesterol Metabolizer® Compare To Other Cholesterol Reducing Formulas?
There are other natural cholesterol reducing products on the market. You are probably interested to know how they compare to Cholesterol Metabolizer®.
Most do not have the same vital endogenous (naturally occurring in the body) ingredients that Cholesterol Metabolizer® contains. Some formulas contain exogenous ingredients (NOT naturally occurring in the body) such as Guggu and Policosanol. Though exogenous ingredients such as these may be of some benefit, exogenous supplements in general will only work for about six months. Beyond that you will not get any additional benefit from continuing to use them.
Why continue to take (and pay) for exogenous ingredients in your cholesterol reducing formula ?
There are some cholesterol reducing formulas that DO contain the same endogenous ingredients that Cholesterol Metabolizer® has... but take a look at the amount of each ingredient. There is no other product out there that has a whopping 300 mg beta sitosterol, 100 mg beta glucan, 50 mg soy isoflavones and 100 mcg of Chromax® per capsule.
CholesterClear — WeKnowCholesterol.com, is a site which pretends to be an impartial product review site. In actuality, they bad mouth all products except for one (CholesterClear) and they do not link to any other sites except for that one. That is not exacly impartial. In fact it is just a trick to promote "CholesterClear". Enough about their trick . . . let's look at their product - "CholesterClear".
They claim that CholesterClear. is better than "Cholesterol Metabolizer®" because it provides 1200 mg of Beta sitosterol. The point they neglect to mention is that there are no studies whatsoever which even suggest that taking 1200 mg/day of beta sitosterol provides any benefit compared to taking 600mg/day. CholesterClear is in fact NOT any better than Cholesterol Metabolizer® . . . just more expensive.
The "CholesterClear" website which WeKnowCholesterol.com describes as being "so informative" does not even list the The "CholesterClear" ingredients. Probably because it more than likely does not even contain the very important cholesterol reducing supplement beta glucan.
Both Cholest-Off (formulated by Nature Made) and Cholox (formulated by ProCaps Laboratories) provide beta sitosterol (each with their own proprietary sterol blend), but neither one contains Beta Glucan, Chromax® or Soy Isoflavones. Price: $26.53 (Cholox) $25.95 (Cholest-Off)
Dr. Derrick DeSilva, M.D. & Unimark Health promote a formula on TV called Cholestium. It too DOES contain Beta Sitosterol, but has NO Beta Glucan, Chromax® or Soy Isoflavones. And it DOES contain Guggu, which is a fine supplement but being EXOGENOUS has only short time effects. Price: $19.99
ProgressiveHealth produces a cholesterol reducing formula called Retorol. It contains NO Beta Sitosterol what so ever. It does have Vitamin B3, which some people are allergic to and it also contains Guggu which only has short term effects. Price: $19.95
Healthy Choice Nutritionals- produces Cholesterol Care. It DOES contain Beta Sitosterol but only ½ the amount contained in Cholesterol Metabolizer®. It DOES contain Beta Glucan but only ¼ the amount contained in Cholesterol Metabolizer®. Price: $27.00
Look at any other cholesterol reducing products out there and ask yourself:
Are the ingredients endogenous? or exogenous?
How much of each endogenous ingredient is there per capsule?
How many capsules does each bottle contain?
What is the cost per bottle?
What is the cost per equivalent dosage?
Once you have done the math you will know that there is no other product that comes close in comparison to the long term effectiveness, safety and cost of Cholesterol Metabolizer®.
Each caplet contains:
- 100 mcg of Chromax®
- 300 mg of Beta Sitosterol
- 100 mg of Beta Glucan
- 50 mg of Soy Isoflavones
Other ingredients: Dicalcium Phosphate. Stearic Acid, Magnesium Stearate, Silica, Glaze.
60 caplets per bottle, 2 caplets per day
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